Selective Publications:(*, first author; # Corresponding author)
1: Zhu, F.*, Liu, L.*, Li, J.*, Liu, B., Wang, Q., Jiao R., Xu, Y., Wang, L., Sun, S., Sun, X., Younus, M., Wang, C., Hokfel, T., Zhang, B.#, Gu, H.#, Xu, Z.#, Zhou, Z.#. (2022). Cocaine increases quantal norepinephrine secretion through NET-dependent PKC activation in locus coeruleus neurons. Cell Reports. 40(7), 16 August 2022, 111199. https://doi.org/10.1016/j.celrep.2022.111199.
2: Han, Y*, Cao, R*, Qin, L., Chen, L., Tang, A.,#, Südhof, TC#, Zhang, B#. (2022) Neuroligin-3 confines AMPA-receptors into nanoclusters, thereby controlling synaptic strength at the calyx of Held synapses. Science Advances. 8(24):eabo4173
3: Qin, L., Guo, S., Han, Y., Wang, X., Zhang, B#. (2020) Functional mosaic organization of neuroligins in neuronal circuits. Cellular and Molecular Life Sciences. 77, 3117-3127
4: Zhang, B.#, Gokce, O., Hale, D., Brose, N., Südhof, TC. (2018) Autism-associated Neuroligin-4 mutation selectively impairs glycinergic synaptic transmission in mouse brainstem synapses. Journal of Experimental Medicine. 215 (6): 1543-1553
5: Zhang, B.#, Seigneur, E., Wei, P., Gokce, O., Morgan, J., Südhof, TC. (2017) Developmental plasticity shapes synaptic phenotypes of autism-associated neuroligin-3 mutations in the calyx of Held. Molecular Psychiatry. 22, 1483–1491. 
6: Chanda,S.#, Hale, D., Zhang, B., Wernig, M., Südhof, TC.# (2017). Unique vs. Redundant Functions of Neuroligin Genes in Shaping Excitatory and Inhibitory Synapse Properties. J. Neurosci. 37 (29) 6816-6836
7: Chen, L.*, Jiang, M.*, Zhang, B., Gokce, O., Südhof, TC#, (2017). Conditional Deletion of All Neurexins Defines Diversity of Essential Synaptic Organizer Functions for Neurexins. Neuron, 94(3), 611-625.
8: Jiang, M.*, Polepalli, J.*, Chen, L., Zhang, B., Südhof, TC#, Malenka, R#. (2017) Conditional ablation of neuroligin-1 in CA1 pyramidal neurons blocks LTP by a cell-autonomous NMDA receptor-independent mechanism. Molecular Psychiatry. 22(3):375-383.
9: Zhang, B.#, Südhof, TC. (2016). Neuroligins are selectively essential for NMDA-receptor signaling in cerebellar stellate interneurons. J. Neurosci. 36(35):9070-9083. ; Highlighted in J. Neurosci.
10: Zhang, B.*, Chen, L.*, Maxeiner, S., Lee, S., Gokce, O., Südhof, TC#. (2015) Neuroligins Sculpt Cerebellar Purkinje-Cell Circuits by Differential Control of Distinct Classes of Synapses. Neuron. 87(4):781-96. (Cover Story)